Author

Y. Komohara

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:host-pathogen & immune response· 3 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:DNA methylation· 1

Frequent Co-Authors

A. Furumoto· 1 shared A. Shimizu· 1 shared A. Watanabe· 1 shared D. G. Tenen· 1 shared E. Iwanaga· 1 shared E. Thrash· 1 shared H. Bono· 1 shared H. Gatanaga· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 8 mutual co-author links

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1 shared paperMicrobiology

A. Furumoto

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

CD34-positive monocytes are highly susceptible to HIV-1

N. Takahashi, O. Noyori, Y. Komohara, Y. M. Eltalkhawy, M. Hirayama, R. Yoshida, H. Nakayama, M. J. Kuroda, T. Nomura, H. Ishii, T. Matano, H. Gatanaga, S. Oka, M. Takiguchi, S. Suzu

This study identifies CD34+ monocytes as highly susceptible to HIV-1 infection, showing they harbor more proviral DNA and express higher HIV-1 receptors despite cART.

bioRxiv · Feb 26, 2024 · PreprintImmunology

Analysis of chronic host-aspergilloma interactions using a novel mouse model

R. Hamashima, M. Tashiro, Y. Nakano, H. Namie, Y. Ito, T. Hirayama, K. Takeda, N. Iwanaga, K. Nishi, H. Liu, T. Takazono, T. Tanaka, A. Watanabe, Y. Komohara, A. Furumoto, , K. Izumikawa

This paper develops a novel mouse model of aspergilloma to study host-fungal interactions, revealing Th1/Th17 responses and angiogenesis induced by dead fungal hyphae.

bioRxiv · Oct 6, 2023 · PreprintMicrobiology

Dissecting the cell of origin of aberrant SALL4 expression in myelodysplastic syndrome

H. Tatetsu, M. Watanabe, J. Liu, K. Tokunaga, E. Iwanaga, Y. Komohara, E. Thrash, M. Matsuoka, D. G. Tenen, L. Chai

This study identifies the cellular origin of aberrant SALL4 expression in MDS using mass cytometry, showing it is mainly in HSPC and myeloid lineages, with SALL4/p53 co-expression linked to TP53 mutations.

bioRxiv · Dec 5, 2022Cell Biology

Resistance to chemical carcinogenesis induction via a dampened inflammatory response in naked mole-rats

K. Oka, S. Fujioka, Y. Kawamura, Y. Komohara, T. Chujo, K. Sekiguchi, Y. Yamamura, Y. Oiwa, N. Omamiuda-Ishikawa, S. Komaki, Y. Sutoh, S. Sakurai, K. Tomizawa, H. Bono, A. Shimizu, , K. Miura

This study shows naked mole-rats (NMRs) resist chemical carcinogenesis through reduced inflammatory responses linked to necroptosis deficiency.

bioRxiv · Oct 21, 2021Cancer Biology