Author

Winfried Maerz

Medical University of Graz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Genetic & Genomic Medicine· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:GWAS / association mapping· 2 Assay:GWAS summary statistics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Marcus Edi KleberiD· 3 shared Claes OhlssoniD· 2 shared Eleftheria ZegginiiD· 2 shared George Davey SmithiD· 2 shared George V. DedoussisiD· 2 shared Ingrid GergeiiD· 2 shared Jie ZhengiD· 2 shared Jonathan H. TobiasiD· 2 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersCell Biology

Marcus Edi KleberORCID

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3 papers

Recent·Most cited

The COPI coatomer influences LDL receptor activity, hepatic lipid storage, and apoB secretion

Grigorios Panteloglou, Jérôme Robert, Marieke Smit, Nicolette Huijkman, Niels Kloosterhuis, Christopher S. Law, Brian Woods, Alaa Othman, Marcus Edi Kleber, Graciela Delgado, Patrizia Tarugi, Museer A. Lone, Justina Clarinda Wolters, Antoine Rimbert, Anja Kerksiek, , Arnold von Eckardstein

This study identifies COPI coatomer subunits that regulate LDL receptor activity, apoB secretion, and hepatic lipid storage, with genetic variants linked to elevated LDL-C levels.

Atherosclerosis · Aug 25, 2026Peer-reviewedCell Biology

Lowering of circulating sclerostin may increase risk of atherosclerosis and its risk factors: evidence from a genome-wide association meta-analysis followed by Mendelian randomization

Jie Zheng, Eleanor Wheeler, Maik Pietzner, Till F. M. Andlauer, Michelle S. Yau, April E. Hartley, Ben Michael Brumpton, Humaira Rasheed, John P. Kemp, Monika Frysz, Jamie W. Robinson, Sjur Reppe, Vid Prijatelj, Kaare Morten Gautvik, Louise Falk, , Jonathan H. Tobias

This study uses GWAS and Mendelian randomization to show that lower sclerostin levels increase risk of hypertension, type 2 diabetes, myocardial infarction, and atherogenic lipid profiles.

Arthritis & Rheumatology · Apr 25, 2023Peer-reviewedGenetic & Genomic Medicine

Mendelian Randomization analysis reveals a causal influence of circulating sclerostin levels on bone mineral density and fractures

Jie Zheng, Winfried Maerz, Ingrid Gergei, Marcus Edi Kleber, Christiane Drechsler, Christoph Wanner, Vincent Mathias Brandenburg, Sjur Reppe, Kaare Morten Gautvik, Carolina Medina‐Gómez, Enisa Shevroja, Arthur Gilly, Young‐Chan Park, George V. Dedoussis, Eleftheria Zeggini, , Jonathan H. Tobias

This paper uses Mendelian Randomization to show that higher circulating sclerostin levels causally reduce bone mineral density and increase fracture risk.

Journal of Bone and Mineral Research · Jun 6, 2019Peer-reviewedGenetics