Author

Craig H. Kerr

Stanford MedicineORCID 0000-0002-4793-2855Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 2 Developmental Biology· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:protein structure prediction· 2 Assay:proteomics· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1

Frequent Co-Authors

Maria BarnaiD· 3 shared Davide RuggeroiD· 2 shared Gerald C. TiuiD· 2 shared Kotaro FujiiiD· 2 shared Travis C. Lantz· 2 shared Adele XuiD· 1 shared Alicia SolórzanoiD· 1 shared Alma L. BurlingameiD· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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3 shared papersMolecular Biology

Maria BarnaORCID

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Author profile →

4 papers

Recent·Most cited

RAPIDASH: A tag-free enrichment of ribosome-associated proteins reveals compositional dynamics in embryonic tissues and stimulated macrophages

Teodorus Theo Susanto, Victoria Hung, Andrew G. Levine, Yuxiang Chen, Craig H. Kerr, Yongjin Yoo, Juan A. Osés-Prieto, Lisa Fromm, Zijian Zhang, Travis C. Lantz, Kotaro Fujii, Marius Wernig, Alma L. Burlingame, Davide Ruggero, Maria Barna

RAPIDASH enriches ribosome-associated proteins from diverse samples, revealing their roles in neurodevelopment and immune response.

Molecular Cell · Sep 1, 2024Peer-reviewedMolecular Biology

Combinatorial optimization of mRNA structure, stability, and translation for RNA-based therapeutics

Kathrin Leppek, Gun Woo Byeon, Wipapat Kladwang, Hannah K. Wayment-Steele, Craig H. Kerr, Adele Xu, Do Soon Kim, Ved V. Topkar, Christian A. Choe, Daphna Rothschild, Gerald C. Tiu, Roger Wellington-Oguri, Kotaro Fujii, Eesha Sharma, Andrew M. Watkins, , Rhiju Das

This study develops PERSIST-seq and In-line-seq to optimize mRNA stability and translation, finding that in-cell stability is more critical than ribosome load for protein output.

Nature Communications · Mar 22, 2022Peer-reviewedMolecular Biology

A p53-dependent translational program directs tissue-selective phenotypes in a model of ribosomopathies

Gerald C. Tiu, Craig H. Kerr, Craig M. Forester, Pallavi S. Krishnarao, Hannah D. Rosenblatt, Nitin Raj, Travis C. Lantz, Olena Zhulyn, Margot E. Bowen, Leila Shokat, Laura Donatella Attardi, Davide Ruggero, Maria Barna

This study identifies a p53-dependent translational program that explains tissue-selective phenotypes in ribosomopathies by regulating 4E-BP1 and altering the translatome.

Developmental Cell · Jul 1, 2021Peer-reviewedDevelopmental Biology

Dynamic rewiring of the human interactome by interferon signalling

Craig H. Kerr, Michael A. Skinnider, Angel M. Madero, Daniel D. T. Andrews, Richard Greg Stacey, Queenie W. T. Chan, Nikolay Stoynov, Eric Jan, Leonard J. Foster

This study maps the dynamic changes in the human protein-protein interaction network induced by interferon signaling, revealing novel regulatory mechanisms and functional contexts for interferon-stimulated genes.

bioRxiv · Sep 12, 2019 · PreprintSystems Biology