Author

J. M. Davis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Biochemistry· 1 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 5 Organism:mouse· 4 Assay:bulk RNA-seq· 3 Research Focus:cell type annotation· 3 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

A. V. Misharin· 2 shared D. R. Winter· 2 shared G. S. Budinger· 2 shared H. Abdala-Valencia· 2 shared K. M. Ridge· 2 shared P. A. Reyfman· 2 shared A. Bharat· 1 shared A. C. McQuattie-Pimentel· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersImmunology

A. V. Misharin

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Survival in macrophages induces enhanced virulence in Cryptococcus

J. Nielson, A. J. Jezewski, M. Wellington, J. M. Davis

The paper shows that Cryptococcus cells become more virulent after surviving in macrophages, with increased mannose and chitin exposure on their cell walls.

bioRxiv · Sep 3, 2023 · PreprintMicrobiology

MBNL1 regulates programmed postnatal switching between regenerative and differentiated cardiac states.

L. R. J. Bailey, D. Bugg, I. M. Reichardt, C. D. Ortac, J. Gunaje, R. Johnson, M. J. MacCoss, T. Sakamoto, D. P. Kelly, M. Regnier, J. M. Davis

This study identifies MBNL1 as a regulator of cardiac cell state transitions, showing it controls RNA stability to switch between regenerative and differentiated states.

bioRxiv · Mar 16, 2023Cell Biology

Influenza-induced activation of recruited alveolar macrophages during the early inflammatory phase drives lung injury and lethality

C. M. Koch, K. Anekalla, Y.-S. Hu, J. M. Davis, M. Ciesielski, G. Gadhvi, S.-Y. Chen, M. Turner, Y. Cheng, B. M. Coates, H. Abdala-Valencia, P. A. Reyfman, A. V. Misharin, G. S. Budinger, D. R. Winter, K. M. Ridge

This study identifies vimentin's role in driving pro-inflammatory responses in alveolar macrophages during influenza infection, linking it to lung injury and mortality.

bioRxiv · Jun 8, 2020 · PreprintImmunology

The Aging Microenvironment Shapes Alveolar Macrophage Identity in Aging

A. C. McQuattie-Pimentel, Z. Ren, N. Joshi, S. Watanabe, T. Stoeger, M. Chi, Z. Lu, L. Sichizya, R. Piseaux, C.-I. Chen, S. Soberanes, P. A. Reyfman, J. M. Walter, K. R. Anekalla, J. M. Davis, , G. S. Budinger

This study shows that the aging lung microenvironment shapes alveolar macrophage identity, influencing their phenotype during aging and after injury.

bioRxiv · Jul 29, 2019 · PreprintImmunology

Activity-Based Ratiometric FRET Probe Reveals Oncogene-Driven Changes in Labile Copper Pools Induced by Altered Glutathione Metabolism

C. Y.-S. Chung, J. M. Posimo, S. Lee, T. Tsang, J. M. Davis, D. C. Brady, C. J. Chang

This paper introduces a ratiometric FRET copper probe, FCP-1, to image labile Cu(I) pools in live cells and shows oncogene-driven changes in glutathione metabolism reduce labile copper availability.

bioRxiv · Jun 24, 2019Biochemistry